[1] Songtao Q, Yuntao L, Jun P, Chuanping H, Xiaofeng S. Membranous layers of the pituitary gland:histological anatomic study and related clinical issues[J]. Neurosurgery, 2009, 64(3 Suppl):ons1-9.
[2] Qi S, Lu Y, Pan J, Zhang X, Long H, Fan J. Anatomic relations of the arachnoidea around the pituitary stalk:relevance for surgical removal of craniopharyngiomas[J]. Acta Neurochir (Wien), 2011, 153:785-796.
[3] Lu YT, Qi ST, Xu JM, Pan J, Shi J. A membranous structure separating the adenohypophysis and neurohypophysis:an anatomical study and its clinical application for craniopharyngioma[J]. J Neurosurg Pediatr, 2015, 15:630-637.
[4] Pan J, Qi S, Liu Y, Lu Y, Peng J, Zhang X, Xu Y, Huang GL, Fan J. Growth patterns of craniopharyngiomas:clinical analysis of 226 patients[J]. J Neurosurg Pediatr, 2016, 17:418-433.
[5] Bao Y, Pan J, Qi ST, Lu YT, Peng JX. Origin of craniopharyngiomas:implications for growth pattern, clinical characteristics, and outcomes of tumor recurrence[J]. J Neurosurg, 2016, 125:24-32.
[6] Qi S, Peng J, Pan J, Zhang X, Lu Y, Fan J, Huang G. Growth and weight of children with craniopharyngiomas based on the tumour location and growth pattern[J]. J Clin Neurosci, 2013, 20:1702-1708.
[7] Liu Y, Qi ST, Wang CH, Pan J, Fan J, Peng JX, Zhang X, Bao Y, Liu YW. Pathological relationship between adamantinomatous craniopharyngioma and adjacent structures based on QST classification[J]. J Neuropathol Exp Neurol, 2018, 77:1017-1023.
[8] Qi ST, Peng JX, Pan J, Fan J, Zhang SC, Liu Y, Bao Y, Qiu BH, Wu XY. Hypopituitarism mode in patients with craniopharyngioma in relation to tumor growth pattern[J]. Zhonghua Yi Xue Za Zhi, 2018, 98:19-24.[漆松涛, 彭俊祥, 潘军, 樊俊, 张世超, 刘忆, 包贇, 邱炳辉, 伍学炎. 不同生长方式颅咽管瘤患者垂体功能减退模型[J]. 中华医学杂志, 2018, 98:19-24.]
[9] Nie J, Huang GL, Deng SZ, Bao Y, Liu YW, Feng ZP, Wang CH, Chen M, Qi ST, Pan J. The purine receptor P2X7R regulates the release of pro-inflammatory cytokines in human craniopharyngioma[J]. Endocr Relat Cancer, 2017, 24:287-296.
[10] Feng Z, Ou Y, Zhou M, Wu G, Ma L, Zhang Y, Liu Y, Qi S. Functional ectopic neural lobe increases GAP-43 expression via PI3K/AKT pathways to alleviate central diabetes insipidus after pituitary stalk lesion in rats[J]. Neuroscience Letters, 2018, 673:1-6.
[11] Zhou MF, Feng ZP, Ou YC, Peng JJ, Li K, Gong HD, Qiu BH, Liu YW, Wang YJ, Qi ST. Endoplasmic reticulum stress induces apoptosis of arginine vasopressin neurons in central diabetes insipidus via PI3K/Akt pathway[J]. CNS Neurosci Ther, 2019, 25:562-574. |