| [1] Akman B, Tarhan C, Arat Z, et al. Renin-Angiotensin system polymorphisms: a risk factor for progression to end-stage renal disease in vesicoureteral reflux patients. Ren Fail, 2009, 31(3): 196-200.
[2] 杜明艳，封靖，杜会山，等. ACE、AGT基因联合多态性与高血压合并脑梗塞相关. 基础医学与临床，2011, 31（11）：1189-93 .
[3] Stewart JA, Kampman O, Huuhka M, et al. ACE polymorphism and response to electroconvulsive therapy in major depression. Neurosci Lett. 2009,458(3):122-5.
[4]. Baghai TC, Schule C, Zill P, et al. The angiotensin I converting enzyme insertion/deletion polymorphism influences therapeutic outcome in major depressed women, but not in men. Neurosci Lett, 2004, 363(1):38-42.
[5] Wu Y, Wang X, Shen X, et al. The I/D polymorphism of angiotensin-converting enzyme gene in major depressive disorder and therapeutic outcome: A case-control study and meta-analysis. J Affect Disord, 2012，136(3):971-8.
[6] 尤加永 ，袁勇贵 ，侯钢等，晚发性抑郁症与血管紧张素转化酶基因多态性关系研究. 实用临床医药杂志，2009, 10(13):1-3.
[7] Kramer MS, Cutler N, Feighner J. Distinct mechanism for antidepressant activity by blockade of central substance P receptors. Science, 1998, 281(5383):1640-5.
[8] Kramer MS, Winokur A, Kelsey J. Demonstration of the efficacy and safety of a novel substance P (NK1) receptor antagonist in major depression. Neuropsychopharmacology, 2004, 29(2):385-92.
[9] Arinami, T., Li, L., Mitsushio, H., et al, An insertion/deletion polymorphism in the angiotensin converting enzyme gene is associated with both brain substance P contents and affective disorders. Biological Psychiatry, 1996. 40, 1122-27.
[10] Zubenko GS, Nixon RA. Mood-elevating effect of captopril in depressed patients. Am J Psychiatry, 1984, 141(1):110-11.
[11] Suehiro T, Morita T, Inoue M, et al. Increased amount of the angiotensin- converting enzyme (ACE) mRNA originating from the ACE allele with deletion. Hum Genet, 2004, 115(2):91-96.
[12] Tiret L, Rigat B, Visvikis S, et al. Evidence, from combined segregation and linkage analysis, that a variant of the angiotensin I-converting enzyme (ACE) gene controls plasma ACE levels. Am J Hum Genet, 1992, 51(1): 197-205.
[13] Bosker FJ, Hartman CA, Nolte IM, et al. Poor replication of candidate genes for major depressive disorder using genome-wide association data. Mol Psychiatry, 2011, 16(5):516-32.
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