[1]Neubert E, Senger-Sander SN, Manzke VS, et al. Serum and serum albumin inhibit in vitro formation of neutrophil extracellular traps (NETs)[J].Front Immunol,2019,10:12. doi: 10.3389/fimmu.2019.00012. [2]Kenny EF, Herzig A, Krüger R, et al. Diverse stimuli engage different neutrophil extracellular trap pathways[J].Elife,2017,6:e24437. doi: 10.7554/eLife.24437. [3]Luo L, Zhang S, Wang Y, et al. Proinflammatory role of neutrophil extracellular traps in abdominal sepsis[J].Am J Physiol Lung Cell Mol Physiol,2014,307:L586-L596. [4]Shrestha B, Ito T, Kakuuchi M, et al. Recombinant thrombomodulin suppresses histone-induced neutrophil extracellular trap formation[J].Front Immunol,2019,10:2535. doi: 10.3389/fimmu.2019.02535. [5]Pylaeva E, Bordbari S, Spyra I, et al. Detrimental effect of type I IFNs during acute lung infection with pseudo-monas aeruginosa is mediated through the stimulation of neutrophil NETosis[J].Front Immunol,2019,10:2190. doi: 10.3389/fimmu.2019.02190. [6]Ebrahimi F, Giaglis S, Hahn S, et al. Markers of neutrophil extracellular traps predict adverse outcome in community-acquired pneumonia: secondary analysis of a randomised controlled trial[J].Eur Respir J,2018,51:1701389. doi: 10.1183/13993003.01389-2017. [7]Lee KH, Cavanaugh L, Leung H, et al. Quantification of NETs-associated markers by flow cytometry and serum assays in patients with thrombosis and sepsis[J].Int J Lab Hematol,2018,40:392-399. [8]Liu L, Mao Y, Xu B, et al. Induction of neutrophil extracellular traps during tissue injury: Involvement of STING and Toll-like receptor 9 pathways[J].Cell Prolif,2019,52:e12579. doi: 10.1111/cpr.12579. [9]Song C, Li H, Li Y, et al. NETs promote ALI/ARDS inflammation by regulating alveolar macrophage polarization[J].Exp Cell Res,2019,382:111486. doi: 10.1016/j.yexcr.2019.06.031. [10]McDonald B, Davis RP, Kim SJ, et al. Platelets and neutrophil extracellular traps collaborate to promote intravascular coagulation during sepsis in mice[J]. Blood,2017,129:1357-1367. [11]郑玥,郑行,祝元锋,等.白蛋白对脂多糖诱导中性粒细胞胞外诱捕网生成的抑制作用[J].免疫学杂志,2020,36:397-403. [12]Sondo E, Bertelli R, Pesce E, et al, High-content screening identifies vanilloids as a novel class of inhibitors of NET formation[J].Front Immunol,2019,10:963. doi: 10.3389/fimmu.2019.00963. [13]Deng Q, Pan B, Alam HB, et al. Citrullinated histone H3 as a therapeutic target for endotoxic shock in mice[J].Front Immunol,2020,10:2957. doi: 10.3389/fimmu.2019.02957. [14]Pottecher J, Noll E, Borel M, et al. Protocol for TRAUMADORNASE: a prospective, randomized, multicentre, double-blinded, placebo-controlled clinical trial of aerosolized dornase alfa to reduce the incidence of moderate-to-severe hypoxaemia in ventilated trauma patients[J].Trials,2020,21:274. doi: 10.1186/s13063-020-4141-6. [15]Okeke EB, Louttit C, Fry C, et al. Inhibition of neutrophil elastase prevents neutrophil extracellular trap formation and rescues mice from endotoxic shock[J].Biomaterials,2020,238:119836. doi: 10.1016/j.biomaterials.2020.119836. [16]Pedrazza L, Cunha AA, Luft C, et al. Mesenchymal stem cells improves survival in LPS-induced acute lung injury acting through inhibition of NETs formation[J].J Cell Physiol,2017,232:3552-3564. [17]Neumann A, Papareddy P, Westman J, et al. Immunoregulation of neutrophil extracellular trap formation by endothelial-derived p33 (gC1q Receptor)[J].J Innate Immun,2018,10:30-43. [18]Zlatina K, Lütteke T, Galuska SP. Individual impact of distinct polysialic acid chain lengths on the cytotoxicity of histone H1, H2A, H2B, H3 and H4[J].Polymers (Basel),2017,9:720. doi: 10.3390/polym9120720. [19]Bornhöfft KF, Viergutz T, Kühnle A, et al. Nanoparticles equipped with α2,8-linked sialic acid chains inhibit the release of neutrophil extracellular traps[J].Nanomaterials (Basel),2019,9:610. doi: 10.3390/nano9040610. [20]Zlatina K, Saftenberger M, Kühnle A, et al. Polysialic acid in human plasma can compensate the cytotoxicity of histones[J].Int J Mol Sci,2018,19:1679. doi: 10.3390/ijms19061679. [21]Arroyo R, Khan MA, Echaide M, et al. SP-D attenuates LPS-induced formation of human neutrophil extracellular traps (NETs), protecting pulmonary surfactant inactiva-tion by NETs[J].Commun Biol,2019,2:470. doi: 10.1038/s42003-019-0662-5. [22]Wang YP, Guo Y, Wen PS, et al. Three ingredients of safflower alleviate acute lung injury and inhibit NET release induced by lipopolysaccharide[J].Mediators Inflamm,2020,2020:2720369. doi: 10.1155/2020/2720369. [23]Shi Y, Liu T, Nieman DC, et al. Aerobic exercise attenuates acute lung injury through NET inhibition[J].Front Immunol,2020,11:409. doi: 10.3389/fimmu.2020.00409. |