Basic & Clinical Medicine ›› 2015, Vol. 35 ›› Issue (3): 413-416.
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Abstract: Nitric oxide level of cerebral tissue was "double peaks-like" during cerebral ischemia-reperfusion, and the role of nitric oxide will change depending on its level in vivo. Nitric oxide mediated apoptosis mainly through four signal pathways. Nitric oxide can make related protein s-nitrosylation via molecular modification, further regulate signal pathways and affect apoptosis.
Key words: Nitric oxide, Apoptosis, Cerebral ischemia-reperfusion, Signal pathway, S-nitrosylation
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URL: http://journal11.magtechjournal.com/Jwk_jcyxylc/EN/
http://journal11.magtechjournal.com/Jwk_jcyxylc/EN/Y2015/V35/I3/413