基础医学与临床 ›› 2015, Vol. 35 ›› Issue (3): 413-416.
• 短篇综述 • 上一篇 下一篇
边经纬1,李俊发1,罗艳琳2
收稿日期:
修回日期:
出版日期:
发布日期:
通讯作者:
基金资助:
Received:
Revised:
Online:
Published:
摘要: 脑缺血再灌注过程中, 脑组织一氧化氮(NO)含量呈“双峰样”变化。作用也随其在机体内含量变化具有不同表现;NO主要通过4条信号通路影响细胞凋亡。NO可通过分子修饰使相关蛋白发生s-亚硝基化,调节信号通路,进而影响细胞凋亡。
关键词: 一氧化氮, 细胞凋亡, 脑缺血再灌注, 信号通路, s-亚硝基化
Abstract: Nitric oxide level of cerebral tissue was "double peaks-like" during cerebral ischemia-reperfusion, and the role of nitric oxide will change depending on its level in vivo. Nitric oxide mediated apoptosis mainly through four signal pathways. Nitric oxide can make related protein s-nitrosylation via molecular modification, further regulate signal pathways and affect apoptosis.
Key words: Nitric oxide, Apoptosis, Cerebral ischemia-reperfusion, Signal pathway, S-nitrosylation
边经纬 李俊发 罗艳琳. 脑缺血再灌注中一氧化氮对神经元凋亡的影响[J]. 基础医学与临床, 2015, 35(3): 413-416.
0 / / 推荐
导出引用管理器 EndNote|Reference Manager|ProCite|BibTeX|RefWorks
链接本文: http://journal11.magtechjournal.com/Jwk_jcyxylc/CN/
http://journal11.magtechjournal.com/Jwk_jcyxylc/CN/Y2015/V35/I3/413